Cannabis exposure during adolescence increases the likelihood of developing psychotic disorders and bipolar disorder by approximately 100% in early adulthood, according to longitudinal psychiatric research. The elevated risk appears to persist for several years following initial use, suggesting a critical vulnerability window during brain development. This finding strengthens the mounting evidence that adolescent cannabis use carries distinct neuropsychiatric risks compared to adult consumption. The developing brain's endocannabinoid system undergoes significant maturation during the teenage years, and THC exposure may disrupt normal neural circuit formation in regions governing mood regulation and reality testing. While the absolute risk remains relatively low for most individuals, the doubling represents a substantial population-level concern given widespread adolescent cannabis access. The research adds weight to existing concerns about high-potency cannabis products, which contain THC concentrations far exceeding those studied in earlier generations of research. For families and clinicians, these data underscore the importance of delayed cannabis initiation, particularly for adolescents with family histories of psychiatric disorders. However, the observational nature of most cannabis research makes it challenging to establish definitive causality versus correlation with underlying genetic predispositions to mental health conditions.